This was a bidirectional ventricular tachycardia in the context of digoxin toxicity, which subsequently changed to a monomorphic ventricular tachycardia. Investigations revealed normal renal function and a blood digoxin level of 2.66 ng/mL. Treatment with phenytoin and ant-digoxin antibodies was started, with resolution of the tachycardia (Figure).
Journal Information
Vol. 69. Issue 2.
Pages 215 (February 2016)
Vol. 69. Issue 2.
Pages 215 (February 2016)
ECG Contest
DOI: 10.1016/j.rec.2015.05.022
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Response to ECG, January 2016
Respuesta al ECG de enero de 2016
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Pablo Merás Colunga
Servicio de Cardiología, Hospital Universitario La Paz, Madrid, Spain
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