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Vol. 70. Issue 8.
Pages 668 (August 2017)
Vol. 70. Issue 8.
Pages 668 (August 2017)
ECG Contest
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Response to ECG, July 2017
Respuesta al ECG de julio de 2017
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Moisés Rodríguez-Mañero
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moirmanero@gmail.com

Corresponding author:
, Javier García-Seara, José Luis Martínez-Sande
Unidad de Arritmias, Servizo de Cardioloxía, Complexo Hospitalario Universitario de Santiago de Compostela (CHUS), Santiago de Compostela, A Coruña, Spain
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Rev Esp Cardiol. 2017;70:59310.1016/j.rec.2017.01.017
Moisés Rodríguez-Mañero, Javier García-Seara, José Luis Martínez-Sande
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The Holter recording (Figure) shows several episodes of atrioventricular block (AVB), at times with P-wave block and constant PR interval before and after the blocked impulse. In theory, these observations define Mobitz II second-degree AVB. However, we see that during these episodes there is a progressive slowing of sinus rhythm. Back in 1978, Massie described how slow sinus rhythm interferes with a diagnosis of Mobitz II AVB because the lower rhythm facilitates conduction of impulses along the atrioventricular node and, therefore, negates the suppressive impact of the vagal effect on node conduction (thus, option 1 is incorrect).

Figure
(0.32MB).

This type of vagally-mediated AVB is a type of pseudo-Mobitz II AVB (option 3), which should be differentiated from bradycardia-dependent AVB1 (option 2). The absence of intraventricular block in the baseline ECG, accelerated heart rate after the pause (asterisk), PP interval prolongation at the same time as the P-wave block (arrow), and appearance of the P-wave in the context of vagal discharge help guide diagnosis and rule out an intrinsic problem in the conduction system. This has diagnostic and therapeutic repercussions, given that bradycardia-dependent AVB is located within the His-Purkinje system (therefore, option 4 is incorrect).

Reference
[1]
S.S. Barold, D.L. Hayes.
Second-degree atrioventricular block: a reappraisal.
Mayo Clin Proc., 76 (2001), pp. 44-57
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