Publish in this journal
Journal Information
Vol. 61. Issue 4.
Pages 431-432 (April 2008)
Share
Share
Download PDF
More article options
Vol. 61. Issue 4.
Pages 431-432 (April 2008)
Response
Full text access
Response
Respuesta
Visits
...
Antonio Bayés de Lunaa
a Institut Catal?? Ci??ncies Cardiovasculars (ICCC), Hospital de la Santa Creu i Sant Pau, Barcelona, Spain
Article information
Full Text
Bibliography
Download PDF
Statistics
Full Text

To the Editor:

First of all, I would like to thank Dr García Cosío for his interest in our work on the ECG-MRI correlation,1-3 which we summarized in an editorial published in the Revista Española de Cardiología last October.4 In response to his letter, I would like to make the following comments.

For more than 40 years,5 the true posterior wall of the heart has been considered the portion corresponding to the inferobasal part of the inferior wall. This area was believed to curve upward. An infarction therein causes a necrosis vector running from back to front, which explains the R-wave in V1 that was the key electrocardiographic criterion of posterior infarction. Nonetheless, magnetic resonance imaging has shown that1-3: a) curvature of the inferobasal part of the inferior wall is uncommon (about 20%); b) even though a posterior wall might exist, we have proven that infarction of this area does not originate an R-wave in V1 because the necrosis vector runs toward V3-V4, and not toward V1; c) in contrast, with the same correlation, when necrosis occurs in the anterior or posterior parts of the lateral wall (particularly middle and low), the necrosis vector runs toward V1 and explains the R at this lead; and d) following the work done by Durrer,6 necrosis of the inferobasal part of the inferior wall, classically called the posterior wall, cannot cause Q-wave in the leads on the back or R-wave in V1 because this area depolarizes after 30 to 40 ms. Therefore, infarction of the so-called posterior wall may naturally exist, but does not cause the R-wave in V1 that is observed in some lateral infarctions.

Dr García Cosío states that "computerized navigation reconstruction of the left ventricle allows models of the left ventricle to be created in its true position and shows infarction areas in a posterior position." The area that he marks with the arrows in his Figure 2 as a posterior infarction corresponds to the posterior part of the lateral wall. The true posterior wall, if it exists, would be located to the right of the arrows (segment 4 of the Cerqueira classification6). Often, the entire lateral wall is the part affected when there is R-wave at V1, and not just the posterior part of the wall. What Dr Cosío points out as the posterior wall in his Figure 1 on the left is actually the lateral wall, which is opposite to the septal wall, and what he indicates as the posterior left ventricle on the right side of the figure is the posterior part of this lateral aspect, but not actually a posterior wall. The posterior wall of the heart from the time of Perloff has been considered the basal-most part of the inferior aspect.

In summary, I would like to express appreciation for the letter because it has allowed me to point out several aspects that were perhaps unclear and are summarized below: a) the title may cause confusion if our studies are understood to indicate that there is no posterior myocardial infarction; the fact is that there is no posterior wall—considering as such the part of the inferior wall that curves upward—; if the posterior wall existed and were infarcted, it would not produce high R-wave in V1 for the reasons we explain in our editorial; and b) R-wave in V1 is due to infarction of the lateral wall, the wall opposite the septal wall which, naturally, has an anterior portion and another posterior portion, although R-wave in V1 is often seen when both parts of the lateral wall are necrotic.

Bibliography
[1]
Cino JM, Pujadas S, Carreras F, Cygankiewicz I, Leta R, Noguero M, et al..
Utility of contrast-enhanced cardiovascular magnetic resonance (CE-DMR) to assess how likely is an infarct to produce a typical ECG pattern..
J Cardiovasc Magn Reson, 8 (2006), pp. 335-44
[2]
Bayés de Luna A, Cino JM, Pujadas S, Cygankiewicz I, Carreras F, García-Moll X, et al..
Concordance of electrocardiographic patterns and healed myocardial infarction location detected by cardiovascular magnetic resonance..
Am J Cardiol, 97 (2006), pp. 443-51
[3]
Bayés de Luna A, Wagner G, Birnbaum Y, Nikus K, Fiol M, Gor-gels A, et al..
A new terminology of left ventricular walls and location of myocardial infarcts that present Q wave based on the standard of cardiac magnetic resonance imaging: A statement for healthcare professionals from a Committee appointed by the International Society for Holter and Noninvasive Electrocardiography..
Circulation, 114 (2006), pp. 1755-60
[4]
Bayés de Luna A..
Nueva terminología de las paredes del corazón y nueva clasificación electrocardiográfica de los infartos con onda Q basada en la correlación con la resonancia magnética..
Rev Esp Cardiol, 60 (2007), pp. 683-9
[5]
Perloff J..
The recognition of strictly posterior myocardial infarction by conventional scalar electrocardiography..
Circulation, 30 (1964), pp. 706
[6]
Cerqueira M..
Standardized myocardial segmentation and nomenclature for tomographic imaging of the heart: a statement for healthcare professionals from the Cardiac Imaging Committee of the Council on Clinical Cardiology of the American Heart Association..
Circulation, 105 (2002), pp. 539-42
Idiomas
Revista Española de Cardiología (English Edition)

Subscribe to our newsletter

View newsletter history
Article options
Tools
es en

¿Es usted profesional sanitario apto para prescribir o dispensar medicamentos?

Are you a health professional able to prescribe or dispense drugs?